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Antibody Found Effective against Autoimmune NMO in Preclinical Study

Researchers at the University of Osaka in Japan have demonstrated, in a preclinical study on human autopsy samples and in a disease-relevant rat model, that treatment with a monoclonal antibody against against a glycoprotein reduces the expression of CXCL2 in macrophages. The researchers observed fewer neutrophils at the damaged nerves, less astrocytic death, and decreased movement and pain symptoms associated with neuromyelitis optica.